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Author (up) Andrade-Pacheco, R.; Savory, D.J.; Midekisa, A.; Gething, P.W.; Sturrock, H.J.W.; Bennett, A.
Title Household electricity access in Africa (2000-2013): Closing information gaps with model-based geostatistics Type Journal Article
Year 2019 Publication PloS one Abbreviated Journal PLoS One
Volume 14 Issue 5 Pages e0214635
Keywords Remote Sensing
Abstract Household electricity access data in Africa are scarce, particularly at the subnational level. We followed a model-based Geostatistics approach to produce maps of electricity access between 2000 and 2013 at a 5 km resolution. We collated data from 69 nationally representative household surveys conducted in Africa and incorporated nighttime lights imagery as well as land use and land cover data to produce maps of electricity access between 2000 and 2013. The information produced here can be an aid for understanding of how electricity access has changed in the region during this 14 year period. The resolution and the continental scale makes it possible to combine these data with other sources in applications in the socio-economic field, both at a local or regional level.
Address Malaria Elimination Initiative, Institute for Global Health Sciences, UCSF, San Francisco, CA, United States of America
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Language English Summary Language Original Title
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ISSN 1932-6203 ISBN Medium
Area Expedition Conference
Notes PMID:31042727; PMCID:PMC6493706 Approved no
Call Number GFZ @ kyba @ Serial 2531
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Author (up) Aubé, M.; Roby, J.; Kocifaj, M.
Title Evaluating potential spectral impacts of various artificial lights on melatonin suppression, photosynthesis, and star visibility Type Journal Article
Year 2013 Publication PloS one Abbreviated Journal PLoS One
Volume 8 Issue 7 Pages e67798
Keywords Humans; *Light; Lighting/methods; Melatonin/*metabolism; Photosynthesis/*radiation effects; Plant Development/radiation effects; blue light; circadian disruption
Abstract Artificial light at night can be harmful to the environment, and interferes with fauna and flora, star visibility, and human health. To estimate the relative impact of a lighting device, its radiant power, angular photometry and detailed spectral power distribution have to be considered. In this paper we focus on the spectral power distribution. While specific spectral characteristics can be considered harmful during the night, they can be considered advantageous during the day. As an example, while blue-rich Metal Halide lamps can be problematic for human health, star visibility and vegetation photosynthesis during the night, they can be highly appropriate during the day for plant growth and light therapy. In this paper we propose three new indices to characterize lamp spectra. These indices have been designed to allow a quick estimation of the potential impact of a lamp spectrum on melatonin suppression, photosynthesis, and star visibility. We used these new indices to compare various lighting technologies objectively. We also considered the transformation of such indices according to the propagation of light into the atmosphere as a function of distance to the observer. Among other results, we found that low pressure sodium, phosphor-converted amber light emitting diodes (LED) and LED 2700 K lamps filtered with the new Ledtech's Equilib filter showed a lower or equivalent potential impact on melatonin suppression and star visibility in comparison to high pressure sodium lamps. Low pressure sodium, LED 5000 K-filtered and LED 2700 K-filtered lamps had a lower impact on photosynthesis than did high pressure sodium lamps. Finally, we propose these indices as new standards for the lighting industry to be used in characterizing their lighting technologies. We hope that their use will favor the design of new environmentally and health-friendly lighting technologies.
Address Departement de physique, Cegep de Sherbrooke, Sherbrooke, Quebec, Canada. martin.aube@cegepsherbrooke.qc.ca
Corporate Author Thesis
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Language English Summary Language Original Title
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ISSN 1932-6203 ISBN Medium
Area Expedition Conference
Notes PMID:23861808; PMCID:PMC3702543 Approved no
Call Number IDA @ john @ Serial 282
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Author (up) Barclay, J.L.; Husse, J.; Bode, B.; Naujokat, N.; Meyer-Kovac, J.; Schmid, S.M.; Lehnert, H.; Oster, H.
Title Circadian desynchrony promotes metabolic disruption in a mouse model of shiftwork Type Journal Article
Year 2012 Publication PloS one Abbreviated Journal PLoS One
Volume 7 Issue 5 Pages e37150
Keywords Animals; Biological Clocks/*physiology; Circadian Rhythm/*physiology; Disease Models, Animal; Eating/genetics; Gene Expression Regulation; Liver/metabolism; Male; Mice; Sleep Disorders, Circadian Rhythm/*metabolism/physiopathology; Suprachiasmatic Nucleus/*metabolism; Transcriptome
Abstract Shiftwork is associated with adverse metabolic pathophysiology, and the rising incidence of shiftwork in modern societies is thought to contribute to the worldwide increase in obesity and metabolic syndrome. The underlying mechanisms are largely unknown, but may involve direct physiological effects of nocturnal light exposure, or indirect consequences of perturbed endogenous circadian clocks. This study employs a two-week paradigm in mice to model the early molecular and physiological effects of shiftwork. Two weeks of timed sleep restriction has moderate effects on diurnal activity patterns, feeding behavior, and clock gene regulation in the circadian pacemaker of the suprachiasmatic nucleus. In contrast, microarray analyses reveal global disruption of diurnal liver transcriptome rhythms, enriched for pathways involved in glucose and lipid metabolism and correlating with first indications of altered metabolism. Although altered food timing itself is not sufficient to provoke these effects, stabilizing peripheral clocks by timed food access can restore molecular rhythms and metabolic function under sleep restriction conditions. This study suggests that peripheral circadian desynchrony marks an early event in the metabolic disruption associated with chronic shiftwork. Thus, strengthening the peripheral circadian system by minimizing food intake during night shifts may counteract the adverse physiological consequences frequently observed in human shift workers.
Address Max Planck Institute of Biophysical Chemistry, Gottingen, Germany
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ISSN 1932-6203 ISBN Medium
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Notes PMID:22629359; PMCID:PMC3357388 Approved no
Call Number IDA @ john @ Serial 94
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Author (up) Bijveld, M.M.C.; van Genderen, M.M.; Hoeben, F.P.; Katzin, A.A.; van Nispen, R.M.A.; Riemslag, F.C.C.; Kappers, A.M.L.
Title Assessment of night vision problems in patients with congenital stationary night blindness Type Journal Article
Year 2013 Publication PloS one Abbreviated Journal PLoS One
Volume 8 Issue 5 Pages e62927
Keywords Vision; Adolescent; Adult; Case-Control Studies; Child; *Dark Adaptation; Electroretinography; Eye Diseases, Hereditary/*physiopathology; Female; Genetic Diseases, X-Linked/*physiopathology; Humans; Light; Male; Middle Aged; Myopia/*physiopathology; Night Blindness/*physiopathology; *Night Vision; *Pattern Recognition, Visual; Surveys and Questionnaires; *Visual Acuity; Visual Fields
Abstract Congenital Stationary Night Blindness (CSNB) is a retinal disorder caused by a signal transmission defect between photoreceptors and bipolar cells. CSNB can be subdivided in CSNB2 (rod signal transmission reduced) and CSNB1 (rod signal transmission absent). The present study is the first in which night vision problems are assessed in CSNB patients in a systematic way, with the purpose of improving rehabilitation for these patients. We assessed the night vision problems of 13 CSNB2 patients and 9 CSNB1 patients by means of a questionnaire on low luminance situations. We furthermore investigated their dark adapted visual functions by the Goldmann Weekers dark adaptation curve, a dark adapted static visual field, and a two-dimensional version of the “Light Lab”. In the latter test, a digital image of a living room with objects was projected on a screen. While increasing the luminance of the image, we asked the patients to report on detection and recognition of objects. The questionnaire showed that the CSNB2 patients hardly experienced any night vision problems, while all CSNB1 patients experienced some problems although they generally did not describe them as severe. The three scotopic tests showed minimally to moderately decreased dark adapted visual functions in the CSNB2 patients, with differences between patients. In contrast, the dark adapted visual functions of the CSNB1 patients were more severely affected, but showed almost no differences between patients. The results from the “2D Light Lab” showed that all CSNB1 patients were blind at low intensities (equal to starlight), but quickly regained vision at higher intensities (full moonlight). Just above their dark adapted thresholds both CSNB1 and CSNB2 patients had normal visual fields. From the results we conclude that night vision problems in CSNB, in contrast to what the name suggests, are not conspicuous and generally not disabling.
Address Bartimeus Institute for the Visually Impaired, Zeist, The Netherlands. mbijveld@bartimeus.nl
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ISSN 1932-6203 ISBN Medium
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Notes PMID:23658786; PMCID:PMC3643903 Approved no
Call Number GFZ @ kyba @ Serial 3051
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Author (up) Blask, David E; Dauchy, Robert T; Dauchy, Erin M; Mao, Lulu; Hill, Steven M; Greene, Michael W; Belancio, Victoria P; Sauer, Leonard A; Davidson, Leslie
Title Light exposure at night disrupts host/cancer circadian regulatory dynamics: Impact on the Warburg effect, lipid signaling and tumor growth prevention Type Journal Article
Year 2014 Publication PloS one Abbreviated Journal
Volume 9 Issue 8 Pages e102776
Keywords
Abstract The central circadian clock within the suprachiasmatic nucleus (SCN) plays an important role in temporally organizing and coordinating many of the processes governing cancer cell proliferation and tumor growth in synchrony with the daily light/dark cycle which may contribute to endogenous cancer prevention. Bioenergetic substrates and molecular intermediates required for building tumor biomass each day are derived from both aerobic glycolysis (Warburg effect) and lipid metabolism. Using tissue-isolated human breast cancer xenografts grown in nude rats, we determined that circulating systemic factors in the host and the Warburg effect, linoleic acid uptake/metabolism and growth signaling activities in the tumor are dynamically regulated, coordinated and integrated within circadian time structure over a 24-hour light/dark cycle by SCN-driven nocturnal pineal production of the anticancer hormone melatonin. Dim light at night (LAN)-induced melatonin suppression disrupts this circadian-regulated host/cancer balance among several important cancer preventative signaling mechanisms, leading to hyperglycemia and hyperinsulinemia in the host and runaway aerobic glycolysis, lipid signaling and proliferative activity in the tumor.
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Call Number LoNNe @ schroer @ Serial 1579
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